Before moving to the field of clinical research, my work involved arsenic toxicogenomics and molecular genetics, addressing the issue of arsenic calamity in the Indian subcontinent. My research included genetic mechanisms and factors influencing inter-individual genetic susceptibility toward arsenic toxicity. During my seven years studying arsenic toxicology, I worked on various aspects of genetic and epigenetic regulations of arsenicosis and development of arsenic induced cancers. As a Research Associate after my Ph.D, I studied DNA methylation, epigenetic dysregulation, and miRNA profiling to identify arsenic-induced toxicity and carcinogenicity. My work included studying global and gene specific DNA methylation, epigenetic dysregulation caused by arsenic exposure, and miRNA profiles to see effect of arsenic on the regulation of gene expression networks. Later on I joined the University of Colorado nschutz Medical Campus as a Postdoctoral Fellow where I focused on genetic and epigenetic regulation of the mouse reproductive axis. During my second postdoctoral work at the University of Maryland, I worked on contact ediated cell signaling with special emphasis on live cell transcription imaging and analysis.